Is PKD a terminal illness? PKD is a innate kidney disease caused by genetic
lesions. Patients with PKD experience numerous cysts in their kidney. The cysts
lead to kidney’ size enlarged and renal functions decrease. Some patients’
kidneys may enlarge to twice of normal size due to cysts’ increase. In that way
, Is PKD a terminal illness?
Any genetic disease can not be uprooted at present. But patients with PKD
still can live normally as long as control the disease effectively.
Western medicine generally uses surgery, but it only can ease oppression to
kidney, and has not method to small cysts. This leads the small cysts enlarge
rapidly and oppress kidney again. This process moves in cycles will leads kidney
function lose day by day.
What should patients with PKD do if western medicine is not the best method
to PKD?
If PKD has been in kidney failure, we are not advocate surgery. The surgery
unable stop continuous failure of kidney function, so some patients will
experience symptoms of end-stage uremia. At this time, kidney function has
completely failure and can not filter waste of the body. When patients progress
to urina, their Kidney functions are unable t o reverse.
Kidneys with cysts are unable to absorb traditional Chinese medicine well, so
Micro-Chinese Medicine Osmotherapy is suggested to patients. This therapy is
external use small molecule medicine to increase absorbtance of medicine and
protect kidney functions. In addition, It has a great effect on kidney function
to receive treatment activity when kidney function can be revered.
In view of this, to stop renal function deterioration and improve the
prognosis of PKD,the effective treatment is to shrink the enlarged cysts and
stop the small ones from enlarging.
Micro-Chinese Medicine Osmotherapy is a preferred treatment option for the
patients with PKD. It has been applied for more than 25 years in clinic and
showed enormous treatment effects. This therapy is designed to shrink the large
cysts by inactivating the epithelial cells on cyst wall.On the other hand, the
effective medicines can soften the cyst wall and improve the permeability of
cyst wall to promote the excretion of cystic fluids, thus shrinking the large
ones.
Micro-Chinese Medicine Osmotherapy can treat PKD effectively and stop its
progression. Thereby, the prognosis of PKD will be improved remarkably.
2014年3月21日星期五
2014年3月17日星期一
What foods can Patients With PKD take
Patients with renal insufficiency or uremia which caused by Polycystic Kidney Disease should pay attention to their diet:
Foods which patients can not take or only can take a little:
1. Inebriant: Patients shouldn’t take any liquor, especially white spirit. Liquor may stimulates polycystic activity of the protein, and accelerate growth of cysts.
2. Fermented food: Fermented food mainly means bacterium change and fermentative foods. Such as sufu and rotten eggs. Avoid eating this type of foods can slow down cysts’ growth.
3. High protein foods: Each kidney disease patients should take low protein foods and avoid compounding of nitrogen metabolin to ease kidney excretion, such as soybean, bean curd and other bean foods.
4. Viscera: Cooked creatural viscera are not unfif for patients with PKD, especially creatural liver. Chinese always think what you eat, what it supplements. Actually, this is fault. Many toxines leave in liver or kidney during butcher process, especially liver. Liver function is detoxify, so many toxines leave in liver. If patients take these foods may increased kidney burden and aggravate illness condition.
5. coffee, chocolate
Foods which should eat more:
1. Fruits and vegetables which contain high level vitamin
2. iron-containing affluent fruits and vegetables, such as carrot
3. Vegetables which can flux stone, such as black fungus
4. Foods which has diuretic function
5. Base-forming food also should take more, these have subsidiarity
But, dietary therapy is only a subsidiary method. Patients should receive treatment early. If you experience PKD, you can think about Micro-Chinese Medicine Osmotherapy.
2014年3月15日星期六
How much do you know about Polycystic Kidney Disease
Outline
Polycystic kidney disease (PKD) is a hereditary disorder of renal cyst which causes gradual enlargement of both kidneys. Sometimes it progresses to renal failure. Almost all forms are caused by a familial genetic mutation. Symptoms and signs include pain in flank of abdomen, hematuria, and high blood pressure. Diagnosis is by CT or ultrasonography. Symptomatic treatment is very effective before kidney failure. But when kidney failure, dialysis or transplantation are needed.
Etiology
PKD can be divided into dominant polycystic kidney disease and autosomal recessive polycystic kidney disease.
Autosomal dominant polycystic kidney disease (ADPKD) has an incidence of 1/1000 and accounts for about 5% of patients with end-stage renal disease. Most patients have not clinical manifestations before adulthood, but penetrance of kidney is complete. In contrast, autosomal recessive PKD is rare. Its incidence is 1/10,000. It causes renal failure during childhood frequently.
In 86 to 96% of cases, ADPKD is caused by mutations in the PKD1 gene on chromosome 16, which codes for the protein polycystin 1; the others are caused by mutations in the PKD2 gene on chromosome 4, which codes for polycystin 2.
Symptoms and Signs
ADPKD usually causes no symptoms initially; one half of patients remain asymptomatic, never develop renal insufficiency or failure, and are never diagnosed. Most patients develop symptoms by the end of their 20s.
Symptoms include low-grade flank, abdominal, and lower back pain due to cystic enlargement and symptoms of infection. Acute pain is usually due to hemorrhage into cysts or renal stone.
Fever is common with acute pyelonephritis, and rupture of cysts into the retroperitoneal space may cause a fever that can last for weeks. Hepatic cysts may cause right upper quadrant pain if they enlarge or become infected. Valvular disorders rarely cause symptoms but occasionally cause heart failure.
Symptoms and signs of unruptured cerebral aneurysm can be absent or may include headache, nausea and vomiting, and cranial nerve deficits; these manifestations warrant immediate intervention
Signs are nonspecific and include hematuria and hypertension (each in about 40 to 50%) and, in 20% of patients, proteinuria in the subnephrotic range (< 3.5g/24 h in adults). Anemia is less common than in other types of chronic kidney disease, presumably because erythropoietin production is preserved. In advanced disease, the kidneys may become grossly enlarged and palpable, causing fullness in the upper abdomen and flank.
Treatment
Supportive Measures to Complications
Strict BP control is essential. Typically an ACE inhibitor or angiotensin receptor blocker is used. In addition to controlling BP, these drugs help block angiotensin and aldosterone, growth factors that contribute to renal scarring and loss of renal function. Urinary tract infection should be treated promptly. Hemodialysis, peritoneal dialysis, or kidney transplantation is required in patients who develop chronic renal failure. ADPKD does not recur in grafts. With dialysis, patients with ADPKD maintain higher Hb levels than any other group of patients with renal failure.
Give ACE inhibitors or angiotensin receptor blockers for hypertension and to help prevent renal scarring and dysfunction; treat other complications as they arise, and consider use of tolvaptan.
Traditional Chinese Medicine Treatment
Traditional Chinese medicine treatment is very effective to patients with CRF, especially the patients in early or medium-stage. This treatment can improve clinical symptoms , control the progression of disease, low creatinine and urea nitrogen. To kidney failure which is caused by PKD, we use Cdonopsis pilosula, Atragalus membranaceus, Angelica sinensis. These can romote blood circulation to remove blood stasis and clear away heat and toxic materials. But it has a long period of treatment.
Polycystic kidney disease (PKD) is a hereditary disorder of renal cyst which causes gradual enlargement of both kidneys. Sometimes it progresses to renal failure. Almost all forms are caused by a familial genetic mutation. Symptoms and signs include pain in flank of abdomen, hematuria, and high blood pressure. Diagnosis is by CT or ultrasonography. Symptomatic treatment is very effective before kidney failure. But when kidney failure, dialysis or transplantation are needed.
Etiology
PKD can be divided into dominant polycystic kidney disease and autosomal recessive polycystic kidney disease.
Autosomal dominant polycystic kidney disease (ADPKD) has an incidence of 1/1000 and accounts for about 5% of patients with end-stage renal disease. Most patients have not clinical manifestations before adulthood, but penetrance of kidney is complete. In contrast, autosomal recessive PKD is rare. Its incidence is 1/10,000. It causes renal failure during childhood frequently.
In 86 to 96% of cases, ADPKD is caused by mutations in the PKD1 gene on chromosome 16, which codes for the protein polycystin 1; the others are caused by mutations in the PKD2 gene on chromosome 4, which codes for polycystin 2.
Symptoms and Signs
ADPKD usually causes no symptoms initially; one half of patients remain asymptomatic, never develop renal insufficiency or failure, and are never diagnosed. Most patients develop symptoms by the end of their 20s.
Symptoms include low-grade flank, abdominal, and lower back pain due to cystic enlargement and symptoms of infection. Acute pain is usually due to hemorrhage into cysts or renal stone.
Fever is common with acute pyelonephritis, and rupture of cysts into the retroperitoneal space may cause a fever that can last for weeks. Hepatic cysts may cause right upper quadrant pain if they enlarge or become infected. Valvular disorders rarely cause symptoms but occasionally cause heart failure.
Symptoms and signs of unruptured cerebral aneurysm can be absent or may include headache, nausea and vomiting, and cranial nerve deficits; these manifestations warrant immediate intervention
Signs are nonspecific and include hematuria and hypertension (each in about 40 to 50%) and, in 20% of patients, proteinuria in the subnephrotic range (< 3.5g/24 h in adults). Anemia is less common than in other types of chronic kidney disease, presumably because erythropoietin production is preserved. In advanced disease, the kidneys may become grossly enlarged and palpable, causing fullness in the upper abdomen and flank.
Treatment
Supportive Measures to Complications
Strict BP control is essential. Typically an ACE inhibitor or angiotensin receptor blocker is used. In addition to controlling BP, these drugs help block angiotensin and aldosterone, growth factors that contribute to renal scarring and loss of renal function. Urinary tract infection should be treated promptly. Hemodialysis, peritoneal dialysis, or kidney transplantation is required in patients who develop chronic renal failure. ADPKD does not recur in grafts. With dialysis, patients with ADPKD maintain higher Hb levels than any other group of patients with renal failure.
Give ACE inhibitors or angiotensin receptor blockers for hypertension and to help prevent renal scarring and dysfunction; treat other complications as they arise, and consider use of tolvaptan.
Traditional Chinese Medicine Treatment
Traditional Chinese medicine treatment is very effective to patients with CRF, especially the patients in early or medium-stage. This treatment can improve clinical symptoms , control the progression of disease, low creatinine and urea nitrogen. To kidney failure which is caused by PKD, we use Cdonopsis pilosula, Atragalus membranaceus, Angelica sinensis. These can romote blood circulation to remove blood stasis and clear away heat and toxic materials. But it has a long period of treatment.
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